Monday, December 3, 2012

Dr. Marwan Sabbagh: The Alzheimer's Prevention Cookbook

  • The Alzheimer's Prevention Cookbook

    <a href="http://www.amazon.com/The-Alzheimers-Prevention-Cookbook-Recipes/dp/1607742470">The Alzheimer's Prevention Cookbook</a> by Dr. Marwan Sabbagh and Beau MacMillan. Published by Ten Speed Press. (The following slides were reprinted with permission from The Alzheimer?s Prevention Cookbook: Recipes to Boost Brain Health by Dr. Marwan Sabbagh and Beau MacMillan, copyright ? 2012. Published by Ten Speed Press, an imprint of the Crown Publishing Group)

  • Sweet Peach Smoothie

    The key to this recipe is using a ripe, in-season peach. It?s always good to get to know the produce guys at your local grocery store because they will let you know when peaches are in their prime. Peaches contain numerous nutrients that are good for your body, including niacin, thiamin, potassium, and calcium. They are also high in beta-carotene, which promotes healthy hearts and eyes. The darker the peach?s color, the more vitamin A it has in its pulp. Peaches may also help in maintaining healthy urinary and digestive functions. There?s some evidence that flaxseed oil may help reduce your risk of heart disease, cancer, stroke, and even diabetes.

  • Sweet Peach Smoothie

    MAKES ABOUT 3 CUPS 11/2 cups apple juice 1 ripe peach, peeled, pitted, and chopped (about 3/4 cup) 3/4 ripe banana, peeled and chopped 1 tablespoon vanilla yogurt 6 ice cubes 2 teaspoons honey 2 teaspoons flaxseed oil Combine the apple juice, peach, banana, yogurt, and ice in a blender and puree until smooth. Add the honey and flaxseed oil and puree briefly to incorporate. Pour into glasses and serve right away.

  • Breakfast Fried Rice with Scrambled Eggs

    Think outside the (cereal) box: fried rice is a great way to fuel up with carbs in the morning. With brown rice, lots of fresh vegetables, and a minimal amount of fat, this recipe is a healthy take on fried rice and is high in vitamin B6. <em>Lop chong</em> is a dried, cooked Chinese sausage with a slightly sweet and smoky flavor; it will require a trip to the Asian grocery store, but you can choose to leave it out.

  • Breakfast Fried Rice with Scrambled Eggs

    MAKES 4 SERVINGS Fried Rice 2 tablespoons chopped lop chong (Chinese sausage; optional) 1/4 cup vegetable oil 1 tablespoon chopped garlic 1 tablespoon peeled, chopped fresh ginger 1 green onion, white and green parts, chopped 2 tablespoons diced red onion 1 or 2 leaves baby bok choy, thinly sliced 1/4 cup shredded red cabbage 5 sugar snap peas, cut into thin strip on the diagonal 2 cups cooked and cooled brown rice 4 tablespoons soy sauce 4 tablespoons mirin Eggs and Garnishes 2 large eggs, beaten 1 teaspoon sesame seeds 2 tablespoons toasted cashews, chopped 1 green onion, white and green parts, sliced thin on the diagonal 2 tablespoons chopped fresh cilantro To make the fried rice, in a large wok or large skillet over high heat, fry the lop chong until rendered, less than a minute. Transfer the lop chong to a paper towel?lined plate and discard the fat. Set the wok over high heat and heat until very hot. Add the oil to the wok. Add the garlic, ginger, chopped green onion, red onion, bok choy, cabbage, and snap peas. Cook, stirring, for 1 minute, or until the vegetables have softened and you can smell the ginger. Add the rice and continue to cook, stirring, until everything is coated, 2 to 4 minutes. Add the soy sauce and mirin and toss well. Remove the wok from the heat. To cook the eggs, heat a small nonstick skillet over medium heat. Spray the pan with nonstick cooking spray, and then pour in the beaten eggs. Cook, gently stirring the eggs, until scrambled but still moist. Transfer fried rice to a serving bowl and top with the scrambled eggs. Sprinkle with the sesame seeds, toasted cashews, and diagonally cut green onion and serve right away.

  • Ahi Tuna on Rye with Spinach Pesto Yogurt

    This is not your average tuna sandwich. For one thing, it?s more like tuna tartare on bread. For another, it?s a very brain-healthy meal. Spinach Pesto Yogurt, not mayonnaise, holds the tuna mixture together, which keeps the amount of saturated fat to a minimum. Tuna is an excellent source of omega-3 fatty acids, the pistachios provide vitamin E, and the raisins are a good source of polyphenol antioxidants. Because the tuna is not cooked in this recipe, be sure to purchase sashimi-grade tuna; ask the fishmonger if you aren?t sure about the quality of the tuna on offer at the seafood counter.

  • Ahi Tuna on Rye with Spinach Pesto Yogurt

    MAKES 2 SANDWICHES 8 ounces sashimi-grade ahi tuna, diced small 2 tablespoons Spinach Pesto Yogurt 2 tablespoons golden raisins 1/4 cup shelled unsalted roasted pistachios, chopped Juice of 1/2 lemon 4 slices rye bread, toasted 1/2 cup alfalfa sprouts 1 teaspoon extra-virgin olive oil Spinach Pesto Yogurt Makes about 3 cups 2 cups loosely packed baby spinach 1 cup extra-virgin olive oil 1/4 cup pine nuts 2 tablespoons grated ?Parmigiano-Reggiano cheese 1 clove garlic, peeled Juice of 1 lemon Pinch of salt Pinch of freshly ground black pepper 1 cup low-fat or nonfat plain yogurt In a medium bowl, combine the tuna, Spinach Pesto Yogurt, raisins, pistachios, and lemon juice, and mix well. Lay out two of the bread slices on a work surface and divide the tuna mixture evenly among them. Put the alfalfa sprouts into a small bowl, drizzle with the olive oil, and toss to combine. Top each portion of tuna with half of the alfalfa sprouts and top with the remaining slices of bread. Cut each sandwich in half and serve right away. Combine the spinach, olive oil, pine nuts, cheese, garlic, lemon juice, salt, and pepper in a blender and puree until well blended. Pour the mixture into a bowl, add the yogurt, and whisk until incorporated. Serve slightly chilled. The mixture will keep in an airtight container in the refrigerator for 3 to 4 days.

  • Striped Bass with Golden Tomato ?and Sweet Pepper Stew

  • Striped Bass with Golden Tomato ?and Sweet Pepper Stew

    MAKES 4 SERVINGS Tomato and Sweet ?Pepper Stew 3 tablespoons olive oil 1 tablespoon finely chopped garlic 1 small shallot, finely chopped 1 fennel bulb, diced large 2 yellow bell peppers, seeded and diced large 3 yellow tomatoes, cored and diced large Pinch of saffron threads 2 tablespoons Pernod or other anise liqueur, such as ouzo or anisette 1 cup white wine 2 cups Brain-Boosting Broth Salt and freshly ground black pepper Fish 2 tablespoons olive oil 4 (6-ounce) skin-on striped bass fillets Salt and freshly ground pepper 11/2 tablespoons chopped fresh flat-leaf parsley leaves, for garnish Preheat the oven to 350?F. To make the stew, in a large saucepan over medium heat, warm the olive oil. Add the garlic, shallot, fennel, and bell peppers and cook, stirring occasionally, until the vegetables soften, about ?10 minutes. Add the tomatoes and saffron and continue to cook until the tomatoes soften slightly, about 2 minutes. Pour in the Pernod, bring to a simmer, and cook, stirring, until almost evaporated. Add the white wine, bring to a simmer, and cook until the liquid is reduced by about half, about ?5 minutes. Pour in the broth, season with salt and pepper, and simmer uncovered for 15 minutes to bring the flavors together. While the stew is simmering, cook the fish. In a large ovenproof skillet over medium-high heat, warm the olive oil. Score the fish fillets with a knife and season them with salt and pepper. Add the fish fillets to the skillet skin side down and cook until the skin is golden brown and crisp, about 3 minutes. Slide the skillet into the oven and cook until the fish skin is opaque and flaky, about 5 minutes. Divide the stew among four plates and place a fish fillet on top. Sprinkle with parsley and serve right away. Brain-Boosting Broth Makes 2 quarts 8 quarts water 3 carrots, coarsely chopped 2 white onions, coarsely chopped 2 celery stalks, coarsely chopped 2 bulbs fennel, coarsely chopped 1 parsnip, coarsely chopped 12 cloves garlic, chopped 1/4 cup fresh ginger, peeled and chopped Stems from 1/2 bunch fresh flat-leaf parsley 1 bunch green onions, green and white parts 1 stalk fresh lemongrass, ?cut in half lengthwise 1 tablespoon salt 1 teaspoon black peppercorns 2 cloves 1 teaspoon dried oregano 1 teaspoon dried rosemary 1 bay leaf Combine all of the ingredients in a large stockpot and bring to a boil over high heat. Turn down the heat to maintain a gentle simmer and cook, uncovered, for 2 hours. Strain the broth through a fine-mesh sieve. Use immediately, refrigerate for up to 5 days, or freeze for up to 1 month.

  • Spaghetti Squash with ?Caramelized Onion and Tomato

    Spaghetti squash is an often-overlooked vegetable. But it?s a very powerful ingredient from a brain-health perspective: it?s low in saturated fat, very low in cholesterol, and a good source of niacin, vitamin B6, and pantothenic acid--plus spaghetti squash is a very good source of vitamin C. In this recipe, strands of baked spaghetti squash are the backdrop for sweet caramelized onions that contrast against salty, savory Parmesan cheese. This dish will appeal to adults and kids alike, and it?s a great way to get pasta lovers to eat more vegetables.

  • Spaghetti Squash with ?Caramelized Onion and Tomato

    MAKES 4 SERVINGS Olive oil 1/2 spaghetti squash (about ?2 pounds), seeded 1 tablespoon unsalted butter 1 medium yellow onion, thinly sliced Curry Salt 1/2 cup grated Parmigiano-Reggiano cheese 1/3 cup chopped fresh chives 1 ripe tomato, cored, seeded, and diced Fresh cilantro leaves, for garnish Preheat the oven to 350?F. Drizzle a rimmed baking sheet and the flesh of the squash with a little olive oil. Set the squash half cut side down on the baking sheet. Sprinkle 2 tablespoons of water over the squash and bake, uncovered, until a fork inserted into the thickest part of the flesh meets no resistance, 30 to 45 minutes. Let cool to room temperature. In a large nonstick skillet over medium heat, melt the butter. Add the onion and cook, stirring occasionally, until lightly caramelized, about 5 minutes. Set aside. Using a fork, scrape the squash from the skin into a medium bowl; the flesh will separate into spaghetti-like strands (you should have about ?21/2 cups). Return the skillet with the onion to medium heat and add the squash. Cook, tossing gently, just until heated through, 1 to 2 minutes. Season to taste with Curry Salt; go easy because the cheese will add salt, too. Toss in the cheese and chives and transfer to a serving bowl. Sprinkle with the tomatoes and cilantro and serve right away. Curry Salt Makes about 1/2 cup 1/2 cup fleur de sel 4 teaspoons curry powder Mix the salt and curry powder until well combined. Store in an airtight container in a cool, dark place (the flavor of the salt gets better with age). This recipe will not go bad over time.

  • Curried Quinoa with Green Onions and Basil

  • Curried Quinoa with Green Onions and Basil

    MAKES 4 SERVINGS 11/2 cups Brain-Boosting Broth 3/4 cup quinoa, rinsed 1 teaspoon curry powder 1 teaspoon minced fresh ginger 5 to 7 green onions, white and green parts, chopped 1/4 cup chopped fresh basil leaves 1/4 cup slivered almonds, toasted 1/4 cup dried cherries, chopped Pinch of salt Pinch of freshly ground black pepper Juice of 1 lemon 2 tablespoons extra-virgin olive oil In a medium saucepan over high heat, bring the broth to a boil. Add the quinoa, curry powder, ?and ginger. Turn down the heat to medium-low, cover, and simmer until the quinoa is tender, about 20 minutes. Transfer the quinoa to a rimmed baking sheet, distribute in an even layer, and let cool to room temperature. When cooled, put the quinoa into a medium bowl and add the green onions, basil, almonds, and cherries. Sprinkle with the salt and pepper and toss to combine. Drizzle with the lemon juice and olive oil and toss again. Serve at room temperature or lightly chilled. Brain-Boosting Broth Makes 2 quarts 8 quarts water 3 carrots, coarsely chopped 2 white onions, coarsely chopped 2 celery stalks, coarsely chopped 2 bulbs fennel, coarsely chopped 1 parsnip, coarsely chopped 12 cloves garlic, chopped 1/4 cup fresh ginger, peeled and chopped Stems from 1/2 bunch fresh flat-leaf parsley 1 bunch green onions, green and white parts 1 stalk fresh lemongrass, ?cut in half lengthwise 1 tablespoon salt 1 teaspoon black peppercorns 2 cloves 1 teaspoon dried oregano 1 teaspoon dried rosemary 1 bay leaf Combine all of the ingredients in a large stockpot and bring to a boil over high heat. Turn down the heat to maintain a gentle simmer and cook, uncovered, for 2 hours. Strain the broth through a fine-mesh sieve. Use immediately, refrigerate for up to 5 days, or freeze for up to 1 month.

  • Source: http://www.huffingtonpost.com/dr-marwan-sabbagh/alzheimers-prevention_b_2218864.html

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    Boehner makes counteroffer on debt talks

    WASHINGTON?House Republicans on Monday unveiled a counterproposal to a Democratic plan to avoid the so-called fiscal cliff that includes $800 billion in new government revenue by eliminating write-offs in the tax code while extending current tax rates for all income levels.

    In a letter sent to the White House, Republican leaders outlined a plan that they said would provide $2.2 trillion in deficit reduction over the next decade. On top of the $800 billion in new revenue from a tax code overhaul, Republicans estimated they could save $300 billion by cutting discretionary spending, $600 billion in "health savings," $200 billion in changes to the consumer price index and another $300 billion in mandatory spending.

    The White House last week sent Republicans a proposal that estimated $1.6 trillion in new tax revenue over 10 years and about $400 billion in savings for entitlement programs. It also included new spending for infrastructure projects. Republicans rejected it outright and put forth their own plan on Monday.

    The President
    The White House
    1600 Pennsylvania Avenue, Northwest
    Washington, DC 20500

    Dear Mr. President,

    After a status quo election in which both you and the Republican majority in the House were re-elected, the American people rightly expect both parties to come together on a fair middle ground and address the nation's most pressing challenges.

    To that end, shortly after the election, we presented you with a balanced framework for averting the fiscal cliff by coupling spending cuts and reforms with new tax revenue. We then welcomed Secretary Geithner to the Capitol on November 29 with every expectation that he would lay out a similarly reasonable path.

    Regrettably, the proposal he outlined on behalf of your Administration contains very little in the way of common ground. The proposal calls for $1.6 trillion in new tax revenue, twice the amount you supported during the campaign. The proposal also includes four times as much tax revenue as spending cuts, in stark contrast to the "balanced approach" on which you campaigned. While administration officials are claiming that this proposal contains 2.5 dollars of spending cuts for each dollar in new revenue, counting as part of this ratio previously enacted savings ? as if these were new spending reductions ? only confuses the public debate. What's worse, the modest spending cuts in this offer are cancelled out by the additional 'stimulus' measures the Administration is requesting. And, this proposal would remove any and all limits on federal borrowing.

    We cannot in good conscience agree to this approach, which is neither balanced nor realistic. If we were to take your Administration's proposal at face value, then we would counter with the House-passed Budget Resolution. It assumes an overhaul of our tax code with revenue remaining at historically normal levels and proposes structural reforms to preserve and protect the Nation's entitlement programs, ensuring they are sustainable for the long-term rather than continuing to grow out of control. Some of its key reforms include:

    The House-passed Budget Resolution assumes enactment of structural Medicare reform that offers future beneficiaries guaranteed coverage options, including a traditional fee-for-service Medicare plan. This proposal is based on recent bipartisan efforts and would provide greater support for the poor and the sick and less support for the wealthy. We achieve these reforms in Medicare without affecting current seniors or those nearing retirement. This would slow the projected explosive spending growth in this program and eventually maintain Medicare spending as a share of the economy at 4.75 percent, thus saving the program for future generations.
    The House-passed Budget Resolution reforms Medicaid and provides states with greater flexibility to better deliver health security to beneficiaries, saving the federal government nearly $800 billion over 10 years.
    Separate from savings in our proposal for the 2010 health care law, the House-passed Budget Resolution envisions hundreds of billions in savings in other mandatory spending, including reforms to Federal employee compensation and the Supplemental Nutrition Assistance Program.

    These reforms are, in our view, absolutely essential to addressing the true drivers of our debt, and we will continue to support and advance them. At the same time, mindful of the status quo election and past exchanges on these questions, we recognize it would be counterproductive to publicly or privately propose entitlement reforms that you and the leaders of your party appear unwilling to support in the near-term.

    With the fiscal cliff nearing, our priority remains finding a reasonable solution that can pass both the House and the Senate, and be signed into law in the next couple of weeks. The best way to do this is by learning from and building on the bipartisan discussions that have occurred during this Congress, including the Biden Group, the Joint Select Committee, and our negotiations leading up to the Budget Control Act.

    For instance, on November 1 of last year, Erskine Bowles, the co-chair of your debt commission, presented the Joint Select Committee with a middle ground approach that garnered praise from many fiscal watchdogs and nonpartisan experts. He recommended that both parties agree to a balanced package that includes significant spending cuts as well as $800 billion in new revenue.

    Notably, the new revenue in the Bowles plan would not be achieved through higher tax rates, which we continue to oppose and will not agree to in order to protect small businesses and our economy. Instead, new revenue would be generated through pro-growth tax reform that closes special-interest loopholes and deductions while lowering rates. On the spending side, the Bowles recommendation would cut more than $900 billion in mandatory spending and another $300 billion in discretionary spending. These cuts would be over and above the spending reductions enacted in the Budget Control Act.

    This is by no means an adequate long-term solution, as resolving our long-term fiscal crisis will require fundamental entitlement reform. Indeed, the Bowles plan is exactly the kind of imperfect, but fair middle ground that allows us to avert the fiscal cliff without hurting our economy and destroying jobs. We believe it warrants immediate consideration.

    If you are agreeable to this framework, we are ready and eager to begin discussions about how to structure these reforms so that the American people can be confident that these targets will be reached.

    Again, the American people expect their leaders to find fair middle ground to address the nation's most pressing challenges. To achieve that outcome, we respectfully request that you respond to this letter in a timely fashion and hope that you will refrain from any further action that would undermine good-faith efforts to reach a reasonable and equitable agreement in this critical matter.

    Sincerely,

    John Boehner, Speaker
    Eric Cantor, Majority Leader
    Kevin McCarthy, Majority Whip
    Cathy McMorris Rodgers, Republican Conference Chairman
    Dave Camp, Chairman, Committee on Ways and Means
    Paul Ryan, Committee on the Budget
    Fred Upton, Committee on Energy & Commerce

    Source: http://news.yahoo.com/blogs/ticket/house-republicans-release-counterproposal-fiscal-cliff-talks-204652663--election.html

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    IU Northwest management professor pens parents&#39; guide to ...

    Charles Hobson, Ph.D., addresses serious issue of ?Passing the Trash? in K-12 schools

    http://iun.edu/news/2012/media/PassingTheTrash_BookCover.jpgAround the 10-year anniversary of his first book, ?The Lecherous University: What Every Student and Parent Should Know about the Epidemic of Sexual Harassment on Campus,? lead author Charles Hobson, Ph.D., decided the book?s second edition should include a chapter about the problem in K-12 schools.

    But, when the Indiana University Northwest Professor of Management set out to research the topic, he said, the information he discovered was so unspeakably shocking that the topic begged a book all its own.

    ?Doing the background research for what I was planning to have as just a chapter,? Hobson said, ?I ran into some statistics that I had never seen or heard before that were so shocking, I said, ?Somebody has to do something about this.??

    The resulting book, ?Passing the Trash: A Parent?s Guide to Combat Sexual Abuse/Harassment of Their Children in School,? published by CreateSpace in the fall of 2012, educates readers about the alarming frequency of sexual abuse and harassment in K-12 education and provides advice to help parents protect their children and hold the appropriate leaders and institutions accountable.

    Hobson said three studies prompted his fervor about releasing a book devoted to the problem of sexual abuse and harassment in K-12 education.

    First, a 2004 report by the U.S. Department of Education estimated that nearly 4.5 million (almost 10 percent) of students will have been sexually abused or harassed by an educator sometime between kindergarten and 12th?grade.

    Second, Hobson learned from a 2010 U.S. Government Accountability Office (GAO) report to Congress about the frequency of a silent practice happening in schools known as ?passing the trash,? in which sexual offenders are encouraged to resign voluntarily in exchange for the school providing positive letters of reference and not reporting incidents to police or taking any disciplinary action.

    Finally, Hobson learned from a 2001 national survey by the American Association of University Women that 81 percent of students in grades 8 through 11 had experienced sexual harassment at school and that 38 percent of the respondents said that teachers (and other school employees) sexually harass students.?(more?)

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    About John Laws

    Catholic, married, pro-life, pro-family, parent. Active in the Knights of Columbus. Member of the staff at The Interim, Canada's Life and Family Newspaper.

    Source: http://johnlaws.wordpress.com/2012/12/01/iu-northwest-management-professor-pens-parents-guide-to-combating-sexual-abuse-harassment/

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    Guards Deny Trying to Intimidate Manning

    Bradley Manning's former guards testified today that they did not intimidate the man accused of leaking hundreds of thousands of classified cables to the anti-secrets website Wikileaks during a Jan. 18, 2011 incident that resulted in Manning being placed on a temporary suicide risk watch.

    Manning's attorneys cite the event as a key reason why his pre-trial confinement at the Marine brig in Quantico, Va., was unlawful and warrants the dismissal of the charges against him.

    Manning faces life imprisonment on charges that he leaked the classified military and diplomatic cables to Wikileaks. Details of those charges will come at a trial scheduled for February and are not being discussed at this week's hearing, which is focused on his nine-month confinement at Quantico from July 2010 to April 2011.

    On Jan. 18, 2011 Manning was being moved to his daily "recreation call" in a room at the brig when he experienced an apparent anxiety attack. Manning said Thursday the guards escorting him seemed to have an aggressive attitude that made him feel nervous and ultimately feel faint.

    Manning testified Thursday that he "lost my demeanor" during a later discussion with brig officials about the incident that led them to place him on temporary suicide risk watch.

    Former Marine guards Lance Corporal Joshua Tankersly and Lance Corporal Jonathan Cline testified today that Manning had been moving around while his hand and leg restraints were placed on him for the escort to the exercise room. They said they reminded Manning that he should respond properly to their orders by referring to their ranks when he answered them.

    When Manning entered the recreation room they described a situation in which Manning fell backwards and landed on his backside.

    They then said that when out of his leg restraints Manning ran to a weightlifting machine, hid behind it and began to cry. Both Cline and Tankersly said they could not explain Manning's behavior. Both guards were ordered to leave the room and were replaced by two other guards who escorted Manning back to his cell.

    Cline said he was puzzled when a supervisor later told him "we intimidated him or something like that."

    Each guard said he could not recall if they sounded harsh when they talked to Manning on the way to the exercise room.

    They both said that aside from the January incident, Manning was courteous and professional in his interactions with them. Both described him as an average prisoner, though Tankersly acknowledged that Manning was a high profile detainee who had the attention of high-ranking officials at the base.

    "It's hard to put 'average' on such a high profile, when you have higher ups on base come and check through to that see all was OK," Tankersly said.

    Gunnery Sgt. William Fuller, one of the senior officers at the brig, also testified today about his participation in a Classification and Assessment board that routinely assessed whether Manning's Maximum Custody and Prevention of Injury status should be downgraded. The board never reduced Manning's status during his stay.

    Fuller acknowledged that before the January incident he and another brig official had considered a downgrade because Manning was "doing pretty good."

    He said the Jan. 18incident "kind of reset things ? we had to keep him on Prevention of Injury."

    Fuller also cited Manning's quiet interactions with him as a reason for keeping Manning on that status.

    According to Fuller "he wouldn't communicate ? it seemed like he didn't really want to talk" and that concerned him, given training he had received that being withdrawn could be an indicator of suicidal behavior.

    Fuller admitted that the conversations were really just quick interactions to see how Manning was doing.. When asked to provide examples of longer exchanges he had with other prisoners, Fuller provided brief sentences. That led David Coombs, Manning's defense attorney to say sarcastically, "so if he'd thrown in more words then he would have classified as a Chatty Patty?"

    Manning's attorneys claim that a protest on Jan. 17 by Manning supporters, at the entrance to the base, may have motivated an aggressive attitude towards the detainee.

    Cline recalled other guards "were annoyed" by the protest" because it would close parts of the base and hinder or interrupt how they got home." But Tankersly said the protest had no impact on Manning's treatment.

    Also Read

    Source: http://news.yahoo.com/guards-deny-trying-intimidate-manning-025407484--abc-news-politics.html

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    Sunday, December 2, 2012

    Fitting Extra Electrical Sockets | Safe Home Improvement

    FIT EXTRA ELECTRICAL SOCKETS

    No special manual skills are required, but a sound knowledge of electrical work is an advantage.

    Working with electricity is potentially dangerous, and you should not attempt to convert or add sockets unless you have the necessary experience and knowledge. If in doubt, enlist the help of a professional electrician.

    FITTING-ELECTRICAL-SOCKETS

    FITTING-ELECTRICAL-SOCKETS

    Always turn off the electricity supply at the consumer unit when working on electrical installations. Wear eye protectors and gloves when using a club hammer and bolster.

    An adequate number of electrical sockets will eliminate the inconvenience and expense of extension leads.
    To increase the number of sockets in your home either convert existing single-socket outlets to doubles or fit additional sockets. Single sockets will be either ?flush?, i.e. they don?t stick out from the wall, or ?surface-mounted?, i.e. boxes that do stick out a little bit from the wall.

    2- Preparation
    The following assumes your house is wired conventionally, using modern materials. Old round-pin sockets should be replaced by an electrician.
    Most socket outlets in modern homes are connected to what is known as a ring circuit or ring main. From a 30amp fuse or 32amp miniature circuit breaker (MCB) in the consumer unit, a length of 2.5mm? two-core-and-earth cable runs from one socket to another and then back to the consumer unit where it is connected to the same set of terminals from which it originated.

    There are usually two ring circuits in the average house, one supplies power to the ground floor sockets and the other to the sockets upstairs .At each socket the cable is cut and connected to terminals behind the faceplate.

    Some homes have only one or two socket outlets per room, but the number can be increased with the addition of an extension known as a spur. This comprises a s2.5mm? cable connected to the terminals of an existing socket or to a separate junction box on the ring circuit. From there, the spur cable runs to a new socket. You can have as many spurs as there were socket outlets on the ring originally. Each original double socket counts as two.
    Individual radial circuits are used to supply power to a single
    appliance such as a cooker or shower unit, but radial circuits can also be used to supply a number of sockets. Spurs can also be connected to this multi-outlet type of radial circuit.

    A radial-circuit cable terminates at the last socket ? it does not return to the consumer unit as with a ring-circuit cable. A 2.5mm? two-core-and-earth cable is used for a floor area up to 20m? (24yds?), and the circuit must be protected by a20amp fuse or MCB. Higher-rated fuses and cable are required for larger floor areas.

    Single sockets on a ring or radial circuit can be converted to doubles or even fused triple sockets. Spur sockets can also be upgraded, provided the spur cable is serving only the one socket outlet.

    3 ? Testing the circuit

    Step 1:?Before testing the circuit, turn off the electricity using the main switch on the consumer unit.

    Step 2: Unscrew the socket faceplate and ease it off its mounting box. Note the number of cables feeding the socket ? if three cables are connected, it is safe to upgrade the socket. If two are fitted you should test the circuit.

    Step 3: Disconnect all the appliances on the circuit, then remove the red-colored live wires from the socket terminal and separate these wires. Attach a continuity tester between the two live wires ? if the tester glows, the socket can be changed.

    If the tester fails to light, get a qualified electrician to test the circuit before changing the socket. Similarly, if only one cable is connected to the back
    of your single socket, have that tested by an qualified electrician to make sure it is safe to convert.

    4- Converting ? Surface-mounted sockets
    This is one of the easiest ways to increase the number of socket outlets in your home. No extra wiring is required and little or no redecoration afterwards. Just swap the single mounting box for a double box of the same depth, and connect the new double socket to the existing circuit cable.

    Step 1: Before starting work, turn off the electricity using the main switch on the consumer unit ? check that the poweris off with a proprietary plug-in socket tester.
    Step 2: Remove the two securing screws and pull the old faceplate away from its mounting box. Loosen the terminals securing the wires and remove the faceplate.

    Check that the bare earth wires are covered with green/yellow earth sleeving ? if not, cut a piece of sleeving to length and thread it over the wires, leaving about 12mm (1/2in) of each wire exposed.

    Step 3: Remove the old mounting box. Break the plastic web covering the appropriate ?entry hole? for the cable in the new box. Pass the cable through the hole and screw the box to the wall.

    Use a spirit level to get your mounting boxes horizontal. Slotted holes allow you to adjust the position of each box as you drive the fixing screws into the wall.

    You may be able to use the existing screws and wall plugs. Ifnot, drill new fixing holes in the wall and insert wall plugs.

    ?

    Step 4: Connect the existing wires to the terminals in the new socket ? red to the L-terminal, black to N-terminal and green/yellow to E-terminal. Make sure all the wires are held securely and that no bare wire is exposed.
    When two or more identical wires are connected to a single terminal, they should be twisted together with pliers to ensure a good connection.
    Step 5: Push the socket faceplate into the box, taking care not to trap the wires, then tighten the faceplate screws.
    Step 6: When all is secure, turn the electricity on and test the socket by plugging in a lamp or a plug-in socket tester.

    5- Converting ? Flush sockets
    The simplest way to upgrade a flush socket is to use a special mounting box that converts it to a double surface-mounted socket. The conversion box is attached to the existing metal box, using the two short screws provided.

    Converting a single flush socket to a double or triple flush socket involves more work but the result looks neater. It is probably best to leave this type of conversion until you plan to redecorate.

    Step 1: Having turned off the power at the consumer unit, unscrew and remove the old faceplate and metal mounting box. You may need to cut around the old box in order to release it from the surrounding plaster.
    Step 2: Hold the new metal box in the required position ? this is largely determined by the original hole in the wall, but there is some leeway. Draw round the box to indicate how much masonry needs to be removed.

    Step 3: Using a cordless drill fitted with a masonry bit, drill
    a series of closely-spaced holes in the masonry. Use a depth stop or a piece of tape wrapped around the drill to ensure the holes are all the same depth.

    You can buy a drilling jig for boring out the recesses for mounting boxes ? it is comprised of rows of closely packed tubes mounted on a backing plate that you attach to the wall.

    Step 4: Use a bolster or cold chisel to chop out a neat rectangular recess for the new box. Drill fixing holes and fit wall plugs

    Step 5: Knock out a convenient cable-entry hole in the box and line the hole with a rubber grommet that will prevent the metal chafing the cable. Pass the cable through the hole and screw the box into its recess.
    Use decorator?s filler to make good any damaged plaster.
    Step 6: Connect the existing wires to the terminals in the new socket ? red to the L-terminal, black to N-terminal and green/yellow to E-terminal.
    When two or more identical wires are connected to a single terminal, they should be twisted together with pliers to ensure a good connection.
    Make sure all the wires are held securely and that no bare wire is exposed.

    Dealing with a hollow wall:

    It is relatively easy to convert a flush socket that is set in a plasterboard partition wall.
    Step 1: Turn off the electricity using the main switch on the consumer unit.
    Step 2: Remove the old fitting and draw round the new mounting box. You may need to reposition the box to one side of the original opening to avoid cutting into wooden studs that form the framework of the wall.

    Step 3: Drill holes in the corners of the marked area and cutout the waste with a pad saw.

    Step 4: Punch out the cable-entry hole and fit a grommet in the new box, then feed in the cable. Fit dry-wall flanges to the sides of the box.

    Step 5: Pass the box through the hole in the wall and manoeuvre it until the flanges come up against the inside face of the plasterboard.

    Step 6: Wire the socket as described in Step 6 above and screw it to the mounting box. As you tighten the screws, the flanges grip the plasterboard and hold the box in place.
    Special plastic mounting boxes with hinged flanges are available for fitting sockets to hollow stud partitions.

    ?

    6 ? Fitting extra sockets
    You can add a new socket to the circuit by connecting a length of 2.5mm? two-core-and-earth cable to the terminals of an existing socket, provided that the socket is not on a spur itself or already feeding a spur

    To avoid long cable runs, mount the new socket on the wall directly behind an existing socket in the next room.

    ? If you want a new socket on the opposite side of the room, run the spur cable under the floor. The new cable can rest on the floor or ceiling between joists or it can be secure to the side of a joist with plastic cable clips.

    ? If your new spur has to run across the line of joists, you will have to drill a series of 12mm(1/2in) holes through the joists. These holes must be 50mm (2in) below the tops of the joists to avoid floorboard nails.

    Step 1: Before starting work, turn off the electricity using the main switch on the consumer unit.

    Step 2: Remove the existing socket and box, then drill a hole for a short length of cable through the wall.

    Step 3: Cut a recess for a flush box or fit a surface-mounted box on the other side. Punch out the cable-entry hole for the cable (fit a rubber grommet in a metal box to prevent chafing the cable) and screw the box in place.

    Step 4: Strip a short length of sheathing off both ends of the spur cable, check that the bare earth wires are covered with green/yellow earth sleeving ? if not, cut a piece of sleeving to length and thread it over the wires leaving a12mm (1/2in) of each wire exposed.

    Step 5: Connect the existing wires to the terminals in the new socket ? red to the L-terminal, black to N-terminal and green/yellow to E-terminal.

    When two or more identical wires are connected to a single terminal, they should be twisted together with pliers to ensure a good connection.

    Make sure all the wires are held securely and that no bare wire is exposed.

    At the other end, twist the new wires together with the matching wires from the ring main, and insert them in the terminals of the original socket on the other side of the wall.

    Run longer spur cables along a wall inside plastic mini-trunking and connect them to surface-mounted sockets.

    Whenever you are installing new cable runs, make sure you do not cut the cable too short ? instead push the excess cable back into the wall or below the floor after you have connected the wires to their terminals.

    Feeding a cable behind the skirting can be a problem. Decide where you want to mount the new socket and use a cordless drill fitted with an extra-long masonry bit to cut a channel behind the skirting board. Rake out the debris from behind the skirting with a cold chisel.

    ?

    ?

    ?

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    Source: http://www.safehomeimprovement.com/fitting-extra-electrical-sockets/

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    Friday, November 30, 2012

    ScienceDaily: Gene News

    ScienceDaily: Gene Newshttp://www.sciencedaily.com/news/health_medicine/genes/ Genes and Genetics News. Read today's medical research in genetics including what can damage genes, what can protect them, and more.en-usThu, 29 Nov 2012 01:12:42 ESTThu, 29 Nov 2012 01:12:42 EST60ScienceDaily: Gene Newshttp://www.sciencedaily.com/images/logosmall.gifhttp://www.sciencedaily.com/news/health_medicine/genes/ For more science articles, visit ScienceDaily.Risk of childhood obesity can be predicted at birthhttp://www.sciencedaily.com/releases/2012/11/121128182739.htm A simple formula can predict at birth a baby?s likelihood of becoming obese in childhood, according to a new study.Wed, 28 Nov 2012 18:27:27 ESThttp://www.sciencedaily.com/releases/2012/11/121128182739.htmPonatinib acts against the most resistant types of chronic myeloid leukemiahttp://www.sciencedaily.com/releases/2012/11/121128182719.htm Phase I trial shows third-generation drug helps patients after other treatments fail.Wed, 28 Nov 2012 18:27:27 ESThttp://www.sciencedaily.com/releases/2012/11/121128182719.htmDouble duty: Immune system regulator found to protect brain from effects of strokehttp://www.sciencedaily.com/releases/2012/11/121128143549.htm A small molecule known to regulate white blood cells has a surprising second role in protecting brain cells from the deleterious effects of stroke, researchers report. The molecule, microRNA-223, affects how cells respond to the temporary loss of blood supply brought on by stroke -- and thus the cells' likelihood of suffering permanent damage.Wed, 28 Nov 2012 14:35:35 ESThttp://www.sciencedaily.com/releases/2012/11/121128143549.htmScientists pair blood test and gene sequencing to detect cancerhttp://www.sciencedaily.com/releases/2012/11/121128142651.htm Scientists have combined the ability to detect cancer DNA in the blood with genome sequencing technology in a test that could be used to screen for cancers, monitor cancer patients for recurrence and find residual cancer left after surgery.Wed, 28 Nov 2012 14:26:26 ESThttp://www.sciencedaily.com/releases/2012/11/121128142651.htmResearchers increase understanding of genetic risk factor for type 1 diabeteshttp://www.sciencedaily.com/releases/2012/11/121128132355.htm Researchers have demonstrated how a genetic variant associated with type 1 diabetes and other autoimmune diseases influences susceptibility to autoimmunity.Wed, 28 Nov 2012 13:23:23 ESThttp://www.sciencedaily.com/releases/2012/11/121128132355.htmHuman genetic variation recent, varies among populationshttp://www.sciencedaily.com/releases/2012/11/121128132259.htm Nearly three-quarters of mutations in genes that code for proteins -- the workhorses of the cell -- occurred within the past 5,000 to 10,000 years, fairly recently in evolutionary terms, said genomic and genetic experts.Wed, 28 Nov 2012 13:22:22 ESThttp://www.sciencedaily.com/releases/2012/11/121128132259.htmScientists uncover a novel cooperative effort to stop cancer spreadhttp://www.sciencedaily.com/releases/2012/11/121128122041.htm Scientists have uncovered a group of what have been considered relatively minor regulators in the body that band together to suppress the spread of cancer from its primary site.Wed, 28 Nov 2012 12:20:20 ESThttp://www.sciencedaily.com/releases/2012/11/121128122041.htmChanges in nerve cells may contribute to the development of mental illnesshttp://www.sciencedaily.com/releases/2012/11/121128122035.htm Reduced production of myelin, a type of protective nerve fiber that is lost in diseases like multiple sclerosis, may also play a role in the development of mental illness, according to new research.Wed, 28 Nov 2012 12:20:20 ESThttp://www.sciencedaily.com/releases/2012/11/121128122035.htmFirst success of targeted therapy in most common genetic subtype of non-small cell lung cancerhttp://www.sciencedaily.com/releases/2012/11/121128121505.htm Chemotherapy and a new, targeted therapy work better in combination than chemotherapy alone in treating patients with the most common genetic subtype of lung cancer, new research suggests.Wed, 28 Nov 2012 12:15:15 ESThttp://www.sciencedaily.com/releases/2012/11/121128121505.htmImmune system could play a central role in age-related macular degenerationhttp://www.sciencedaily.com/releases/2012/11/121128093919.htm Changes in how genes in the immune system function may result in age-related macular degeneration (AMD), the leading cause of visual impairment in older adults. The findings are epigenetic in nature, meaning that the underlying DNA is normal but gene expression has been modified, likely by environmental factors, in an adverse way. Environmental factors associated with AMD include smoking, diet, and aging. This is the first epigenetic study revealing the molecular mechanisms for any eye disease.Wed, 28 Nov 2012 09:39:39 ESThttp://www.sciencedaily.com/releases/2012/11/121128093919.htmMany flame retardants in house dust at unsafe levels, study findshttp://www.sciencedaily.com/releases/2012/11/121128093810.htm In a new study of the largest number of flame retardants ever tested in homes, researchers found that most houses had levels of at least one flame retardant that exceeded a federal health guideline.Wed, 28 Nov 2012 09:38:38 ESThttp://www.sciencedaily.com/releases/2012/11/121128093810.htmHow infidelity helps nieces and nephews: Men may share more genes with sisters' kids than cheating wife's kidshttp://www.sciencedaily.com/releases/2012/11/121127190021.htm A new study produced new mathematical support for a theory that explains why men in some cultures often feed and care for their sisters? children: where extramarital sex is common and accepted, a man?s genes are more likely to be passed on by their sister?s kids than by their wife?s kids.Tue, 27 Nov 2012 19:00:00 ESThttp://www.sciencedaily.com/releases/2012/11/121127190021.htmGene linked to respiratory distress in babieshttp://www.sciencedaily.com/releases/2012/11/121127130256.htm Some infants are more susceptible to potentially life-threatening breathing problems after birth, and rare, inherited DNA differences may explain why, according to new research.Tue, 27 Nov 2012 13:02:02 ESThttp://www.sciencedaily.com/releases/2012/11/121127130256.htmNew understanding of X chromosome inactivationhttp://www.sciencedaily.com/releases/2012/11/121127101534.htm Scientists have broadened our understanding of how cells regulate silencing of the X chromosome in a process known as X-inactivation.Tue, 27 Nov 2012 10:15:15 ESThttp://www.sciencedaily.com/releases/2012/11/121127101534.htmNew mechanism for cancer progression discoveredhttp://www.sciencedaily.com/releases/2012/11/121127094311.htm Researchers have discovered an alternative mechanism for activating rhe oncogene Ras that does not require mutation or hormonal stimulus.Tue, 27 Nov 2012 09:43:43 ESThttp://www.sciencedaily.com/releases/2012/11/121127094311.htmProtein injection points to muscular dystrophy treatmenthttp://www.sciencedaily.com/releases/2012/11/121127094248.htm Scientists have discovered that injecting a novel human protein into muscle affected by Duchenne muscular dystrophy significantly increases its size and strength, findings that could lead to a therapy akin to the use of insulin by diabetics.Tue, 27 Nov 2012 09:42:42 ESThttp://www.sciencedaily.com/releases/2012/11/121127094248.htmChemical 'switches' for neurodegenerative diseases discoveredhttp://www.sciencedaily.com/releases/2012/11/121127093951.htm Researchers have identified and ?switched off? a chemical chain that causes neurodegenerative diseases such as Huntington?s disease, amyotrophic lateral sclerosis and dementia. The findings could one day be of particular therapeutic benefit to Huntington?s disease patients.Tue, 27 Nov 2012 09:39:39 ESThttp://www.sciencedaily.com/releases/2012/11/121127093951.htmGene that causes tumor disorder linked to increased breast cancer riskhttp://www.sciencedaily.com/releases/2012/11/121127093855.htm New research showing a more than four-fold increase in the incidence of breast cancer in women with neurofibromatosis-1 (NF1) adds to growing evidence that women with this rare genetic disorder may benefit from early breast cancer screening with mammograms beginning at age 40, and manual breast exams as early as adolescence.Tue, 27 Nov 2012 09:38:38 ESThttp://www.sciencedaily.com/releases/2012/11/121127093855.htmMetabolic protein launches sugar feast that nurtures brain tumorshttp://www.sciencedaily.com/releases/2012/11/121126164003.htm PKM2 slips into nucleus to promote cancer; potential biomarker and drug approach discovered.Mon, 26 Nov 2012 16:40:40 ESThttp://www.sciencedaily.com/releases/2012/11/121126164003.htmPossible new treatment for Ewing sarcomahttp://www.sciencedaily.com/releases/2012/11/121126142855.htm Discovery of a new drug with high potential to treat Ewing sarcoma, an often deadly cancer of children and young adults, and the previously unknown mechanism behind it, come hand-in-hand in a new study.Mon, 26 Nov 2012 14:28:28 ESThttp://www.sciencedaily.com/releases/2012/11/121126142855.htmSurvival gene may be key to controlling HIV and hepatitishttp://www.sciencedaily.com/releases/2012/11/121126131349.htm A newly discovered gene that is essential for embryo survival could also hold the key to treating and potentially controlling chronic infections such as HIV, hepatitis and tuberculosis. The gene, called Arih2, is fundamental to the function of the immune system -- making critical decisions about whether to switch on the immune response to an infection.Mon, 26 Nov 2012 13:13:13 ESThttp://www.sciencedaily.com/releases/2012/11/121126131349.htmMicrobial 'missing link' discovered after man impales hand on tree branchhttp://www.sciencedaily.com/releases/2012/11/121126110737.htm Two years ago, a 71-year-old Indiana man impaled his hand on a branch after cutting down a dead tree. The wound caused an infection that led scientists to discover a new bacterium and solve a mystery about how bacteria came to live inside insects.Mon, 26 Nov 2012 11:07:07 ESThttp://www.sciencedaily.com/releases/2012/11/121126110737.htmTransposable elements reveal a stem cell specific class of long noncoding RNAshttp://www.sciencedaily.com/releases/2012/11/121125192838.htm Over a decade after sequencing the human genome, it has now become clear that the genome is not mostly ?junk? as previously thought. In fact, the ENCODE project consortium of dozens of labs and petabytes of data have determined that these ?noncoding? regions house everything from disease trait loci to important regulatory signals, all the way through to new types of RNA-based genes.Sun, 25 Nov 2012 19:28:28 ESThttp://www.sciencedaily.com/releases/2012/11/121125192838.htmNew molecular culprit linked to breast cancer progressionhttp://www.sciencedaily.com/releases/2012/11/121124090511.htm Researchers have uncovered a protein ?partner? commonly used by breast cancer cells to unlock genes needed for spreading the disease around the body. A report on the discovery details how some tumors get the tools they need to metastasize.Sat, 24 Nov 2012 09:05:05 ESThttp://www.sciencedaily.com/releases/2012/11/121124090511.htmNew insights into virus proteome: Unknown proteins of the herpesvirus discoveredhttp://www.sciencedaily.com/releases/2012/11/121123092132.htm The genome encodes the complete information needed by an organism, including that required for protein production. Viruses, which are up to a thousand times smaller than human cells, have considerably smaller genomes. Using a type of herpesvirus as a model system scientists have shown that the genome of this virus contains much more information than previously assumed. The researchers identified several hundred novel proteins, many of which were surprisingly small.Fri, 23 Nov 2012 09:21:21 ESThttp://www.sciencedaily.com/releases/2012/11/121123092132.htmScientists describe elusive replication machinery of flu viruseshttp://www.sciencedaily.com/releases/2012/11/121122152928.htm Scientists have made a major advance in understanding how flu viruses replicate within infected cells. The researchers used cutting-edge molecular biology and electron-microscopy techniques to ?see? one of influenza?s essential protein complexes in unprecedented detail. The images generated in the study show flu virus proteins in the act of self-replication, highlighting the virus?s vulnerabilities that are sure to be of interest to drug developers.Thu, 22 Nov 2012 15:29:29 ESThttp://www.sciencedaily.com/releases/2012/11/121122152928.htmProtein folding: Look back on scientific advances made as result of 50-year old puzzlehttp://www.sciencedaily.com/releases/2012/11/121122152910.htm Fifty years after scientists first posed a question about protein folding, the search for answers has led to the creation of a full-fledged field of research that led to major advances in supercomputers, new materials and drug discovery, and shaped our understanding of the basic processes of life, including so-called "protein-folding diseases" such as Alzheimer's, Parkinson's and type II diabetes.Thu, 22 Nov 2012 15:29:29 ESThttp://www.sciencedaily.com/releases/2012/11/121122152910.htmStep forward in regenerating and repairing damaged nerve cellshttp://www.sciencedaily.com/releases/2012/11/121121145638.htm Researchers recently uncovered a nerve cell's internal clock, used during embryonic development. This breakthrough could lead to the development of new tools to repair and regenerate nerve cells following injuries to the central nervous system.Wed, 21 Nov 2012 14:56:56 ESThttp://www.sciencedaily.com/releases/2012/11/121121145638.htmArchitecture of rod sensory cilium disrupted by mutationhttp://www.sciencedaily.com/releases/2012/11/121121145621.htm Using a new technique called cryo-electron tomography, scientists have created a three-dimensional map that gives a better understanding of how the architecture of the rod sensory cilium (part of one type of photoreceptor in the eye) is changed by genetic mutation and how that affects its ability to transport proteins as part of the light-sensing process.Wed, 21 Nov 2012 14:56:56 ESThttp://www.sciencedaily.com/releases/2012/11/121121145621.htmAging: Scientists further unravel telomere biologyhttp://www.sciencedaily.com/releases/2012/11/121121130933.htm Researchers have resolved the structure of that allows a telomere-related protein, Cdc13, to form dimers in yeast. Mutations in this region of Cdc13 put the kibosh on the ability of telomerase and other proteins to maintain telomeres.Wed, 21 Nov 2012 13:09:09 ESThttp://www.sciencedaily.com/releases/2012/11/121121130933.htmDrug resistance biomarker could improve cancer treatmenthttp://www.sciencedaily.com/releases/2012/11/121121130811.htm Cancer therapies often have short-lived benefits due to the emergence of genetic mutations that cause drug resistance. A key gene that determines resistance to a range of cancer drugs has been reported in a new study. The study reveals a biomarker that can predict responses to cancer drugs and offers a strategy to treat drug-resistant tumors based on their genetic signature.Wed, 21 Nov 2012 13:08:08 ESThttp://www.sciencedaily.com/releases/2012/11/121121130811.htmGenome packaging: Key to breast cancer developementhttp://www.sciencedaily.com/releases/2012/11/121121130703.htm Two recent studies delve into the role of chromatin modifying enzymes and transcription factors in tumour cells. In one, it was found that the PARP1 enzyme activated by kinase CDK2 is necessary to induce the genes responsible for the proliferation of breast cancer cells in response to progesterone. In another, extensive work has been undertaken to identify those genes activated by the administration of progesterone in breast cancer, the sequences that can be recognized and how these genes are induced.Wed, 21 Nov 2012 13:07:07 ESThttp://www.sciencedaily.com/releases/2012/11/121121130703.htmShort DNA strands in genome may be key to understanding human cognition and diseaseshttp://www.sciencedaily.com/releases/2012/11/121121130643.htm Previously discarded, human-specific ?junk? DNA represents untapped resource in the study of diseases like Alzheimer?s and autism.Wed, 21 Nov 2012 13:06:06 ESThttp://www.sciencedaily.com/releases/2012/11/121121130643.htmBiomarking time: Methylome modifications offer new measure of our 'biological' agehttp://www.sciencedaily.com/releases/2012/11/121121130633.htm In a new study, researchers describe markers and a model that quantify how aging occurs at the level of genes and molecules, providing not just a more precise way to determine how old someone is, but also perhaps anticipate or treat ailments and diseases that come with the passage of time.Wed, 21 Nov 2012 13:06:06 ESThttp://www.sciencedaily.com/releases/2012/11/121121130633.htmKidney tumors have a mind of their ownhttp://www.sciencedaily.com/releases/2012/11/121121104552.htm New research has found there are several different ways that kidney tumors can achieve the same result -- namely, grow.Wed, 21 Nov 2012 10:45:45 ESThttp://www.sciencedaily.com/releases/2012/11/121121104552.htmMechanism to repair clumped proteins explainedhttp://www.sciencedaily.com/releases/2012/11/121121104416.htm Clumped proteins can be dissolved with the aid of cellular repair systems -- a process of critical importance for cell survival especially under conditions of stress. Researchers have now decrypted the fundamental mechanism for dissolving protein aggregates that involves specific molecular chaperones.Wed, 21 Nov 2012 10:44:44 ESThttp://www.sciencedaily.com/releases/2012/11/121121104416.htmNovel mechanism through which normal stromal cells become cancer-promoting stromal cells identifiedhttp://www.sciencedaily.com/releases/2012/11/121121104401.htm New understanding of molecular changes that convert harmless cells surrounding ovarian cancer cells into cells that promote tumor growth and metastasis provides potential new therapeutic targets for this deadly disease, according to new research.Wed, 21 Nov 2012 10:44:44 ESThttp://www.sciencedaily.com/releases/2012/11/121121104401.htmNew test for tuberculosis could improve treatment, prevent deaths in Southern Africahttp://www.sciencedaily.com/releases/2012/11/121120194932.htm A new rapid test for tuberculosis (TB) could substantially and cost-effectively reduce TB deaths and improve treatment in southern Africa -- a region where both HIV and tuberculosis are common.Tue, 20 Nov 2012 19:49:49 ESThttp://www.sciencedaily.com/releases/2012/11/121120194932.htmEvolution of human intellect: Human-specific regulation of neuronal geneshttp://www.sciencedaily.com/releases/2012/11/121120194926.htm A new study has identified hundreds of small regions of the genome that appear to be uniquely regulated in human neurons. These regulatory differences distinguish us from other primates, including monkeys and apes, and as neurons are at the core of our unique cognitive abilities, these features may ultimately hold the key to our intellectual prowess (and also to our potential vulnerability to a wide range of 'human-specific' diseases from autism to Alzheimer's).Tue, 20 Nov 2012 19:49:49 ESThttp://www.sciencedaily.com/releases/2012/11/121120194926.htmRibosome regulates viral protein synthesis, revealing potential therapeutic targethttp://www.sciencedaily.com/releases/2012/11/121120132906.htm Rather than target RNA viruses directly, aiming at the host cells they invade could hold promise, but any such strategy would have to be harmless to the host. Now, a surprising discovery made in ribosomes may point the way to fighting fatal viral infections such as rabies.Tue, 20 Nov 2012 13:29:29 ESThttp://www.sciencedaily.com/releases/2012/11/121120132906.htmHow does antibiotic resistance spread? Scientists find answers in the nosehttp://www.sciencedaily.com/releases/2012/11/121120121835.htm Microbiologists studying bacterial colonization in mice have discovered how the very rapid and efficient spread of antibiotic resistance works in the respiratory pathogen, Streptococcus pneumoniae (also known as the pneumococcus). The team found that resistance stems from the transfer of DNA between bacterial strains in biofilms in the nasopharynx, the area just behind the nose.Tue, 20 Nov 2012 12:18:18 ESThttp://www.sciencedaily.com/releases/2012/11/121120121835.htmScientists identify inhibitor of myelin formation in central nervous systemhttp://www.sciencedaily.com/releases/2012/11/121120100155.htm Scientists have discovered another molecule that plays an important role in regulating myelin formation in the central nervous system. Myelin promotes the conduction of nerve cell impulses by forming a sheath around their projections, the so-called axons, at specific locations -- acting like the plastic insulation around a power cord.Tue, 20 Nov 2012 10:01:01 ESThttp://www.sciencedaily.com/releases/2012/11/121120100155.htm'Obese but happy gene' challenges the common perception of link between depression and obesityhttp://www.sciencedaily.com/releases/2012/11/121120084725.htm Researchers have discovered new genetic evidence about why some people are happier than others. The scientists have uncovered evidence that the gene FTO -- the major genetic contributor to obesity -- is associated with an eight per cent reduction in the risk of depression. In other words, it's not just an obesity gene but a "happy gene" as well.Tue, 20 Nov 2012 08:47:47 ESThttp://www.sciencedaily.com/releases/2012/11/121120084725.htmTelomere lengths predict life expectancy in the wild, research showshttp://www.sciencedaily.com/releases/2012/11/121119213144.htm Researchers have found that biological age and life expectancy can be predicted by measuring an individual's DNA. They studied the length of chromosome caps -- known as telomeres -- in a 320-strong wild population of Seychelles Warblers on a small isolated island.Mon, 19 Nov 2012 21:31:31 ESThttp://www.sciencedaily.com/releases/2012/11/121119213144.htmCancer: Some cells don't know when to stophttp://www.sciencedaily.com/releases/2012/11/121119171403.htm Certain mutated cells keep trying to replicate their DNA -- with disastrous results -- even after medications rob them of the raw materials to do so, according to new research.Mon, 19 Nov 2012 17:14:14 ESThttp://www.sciencedaily.com/releases/2012/11/121119171403.htmMultiple sclerosis ?immune exchange? between brain and blood is uncoveredhttp://www.sciencedaily.com/releases/2012/11/121119163301.htm DNA sequences obtained from a handful of patients with multiple sclerosis have revealed the existence of an ?immune exchange? that allows the disease-causing cells to move in and out of the brain.Mon, 19 Nov 2012 16:33:33 ESThttp://www.sciencedaily.com/releases/2012/11/121119163301.htmFruit fly studies guide investigators to molecular mechanism frequently misregulated in human cancershttp://www.sciencedaily.com/releases/2012/11/121119132056.htm Changes in how DNA interacts with histones ?- the proteins that package DNA ?- regulate many fundamental cell activities from stem cells maturing into a specific body cell type or blood cells becoming leukemic. These interactions are governed by a biochemical tug of war between repressors and activators, which chemically modify histones signaling them to clamp down tighter on DNA or move aside and allow a gene to be expressed.Mon, 19 Nov 2012 13:20:20 ESThttp://www.sciencedaily.com/releases/2012/11/121119132056.htm3-D light switch for the brain: Device may help treat Parkinson's, epilepsy; aid understanding of consciousnesshttp://www.sciencedaily.com/releases/2012/11/121119114249.htm A new tool for neuroscientists delivers a thousand pinpricks of light to individual neurons in the brain. The new 3-D "light switch", created by biologists and engineers, could one day be used as a neural prosthesis that could treat conditions such as Parkinson's and epilepsy by using gene therapy to turn individual brain cells on and off with light.Mon, 19 Nov 2012 11:42:42 ESThttp://www.sciencedaily.com/releases/2012/11/121119114249.htmNew factor of genetic susceptibility to Alzheimer's diseasehttp://www.sciencedaily.com/releases/2012/11/121119104944.htm A large-scale international study has just discovered a gene for susceptibility to a rare disease providing evidence of the heterogeneous aetiology of Alzheimer's disease.Mon, 19 Nov 2012 10:49:49 ESThttp://www.sciencedaily.com/releases/2012/11/121119104944.htmBlood cancer gene BCL6 identified as a key factor for differentiation of nerve cells of cerebral cortexhttp://www.sciencedaily.com/releases/2012/11/121119093848.htm The cerebral cortex is the most complex structure in our brain and the seat of consciousness, emotion, motor control and language. In order to fulfill these functions, it is composed of a diverse array of nerve cells, called cortical neurons, which are affected by many neurological and neuropsychiatric diseases. Researchers have opened new perspectives on brain development and stem cell neurobiology by discovering a gene called BCL6 as a key factor in the generation of cortical neurons during embryonic brain development.Mon, 19 Nov 2012 09:38:38 ESThttp://www.sciencedaily.com/releases/2012/11/121119093848.htmMinority report: Insight into subtle genomic differences among our own cellshttp://www.sciencedaily.com/releases/2012/11/121118141530.htm Scientists have demonstrated that induced pluripotent stem cells -- the embryonic-stem-cell look-alikes whose discovery a few years ago won this year's Nobel Prize in medicine -- are not as genetically unstable as was thought.Sun, 18 Nov 2012 14:15:15 ESThttp://www.sciencedaily.com/releases/2012/11/121118141530.htmSkin cells reveal DNA's genetic mosaichttp://www.sciencedaily.com/releases/2012/11/121118141524.htm The prevailing wisdom has been that every cell in the body contains identical DNA. However, a new study of stem cells derived from the skin has found that genetic variations are widespread in the body's tissues, a finding with profound implications for genetic screening.Sun, 18 Nov 2012 14:15:15 ESThttp://www.sciencedaily.com/releases/2012/11/121118141524.htmLikely basis of birth defect causing premature skull closure in infants identifiedhttp://www.sciencedaily.com/releases/2012/11/121118141432.htm Geneticists, pediatricians, surgeons and epidemiologists have identified two areas of the human genome associated with the most common form of non-syndromic craniosynostosis premature closure of the bony plates of the skull.Sun, 18 Nov 2012 14:14:14 ESThttp://www.sciencedaily.com/releases/2012/11/121118141432.htmDNA packaging discovery reveals principles by which CRC mutations may cause cancerhttp://www.sciencedaily.com/releases/2012/11/121117184658.htm A new discovery concerning a fundamental understanding about how DNA works will produce a "180-degree change in focus" for researchers who study how gene packaging regulates gene activity, including genes that cause cancer and other diseases.Sat, 17 Nov 2012 18:46:46 ESThttp://www.sciencedaily.com/releases/2012/11/121117184658.htmHepatitis C treatment's side effects can now be studied in the labhttp://www.sciencedaily.com/releases/2012/11/121116161059.htm Adverse side effects of certain hepatitis C medications can now be replicated in the lab, thanks to a research team. The new method aids understanding of recent failures of hepatitis C antiviral drugs in some patients, and could help to identify medications that eliminate adverse effects. The findings may aid the development of safer and more effective treatments for hepatitis C and other pathogens such as SARS and West Nile virus.Fri, 16 Nov 2012 16:10:10 ESThttp://www.sciencedaily.com/releases/2012/11/121116161059.htmReconsidering cancer's bad guyhttp://www.sciencedaily.com/releases/2012/11/121116124644.htm Researchers have found that a protein, known for causing cancer cells to spread around the body, is also one of the molecules that trigger repair processes in the brain.Fri, 16 Nov 2012 12:46:46 ESThttp://www.sciencedaily.com/releases/2012/11/121116124644.htmGene distinguishes early birds from night owls and helps predict time of deathhttp://www.sciencedaily.com/releases/2012/11/121116124551.htm New research shows that a gene is responsible for a person's tendency to be an early riser or night owl -- and helps determine the time of day a person is most likely to die.Fri, 16 Nov 2012 12:45:45 ESThttp://www.sciencedaily.com/releases/2012/11/121116124551.htmClues to cause of kids' brain tumorshttp://www.sciencedaily.com/releases/2012/11/121116091226.htm Insights from a genetic condition that causes brain cancer are helping scientists better understand the most common type of brain tumor in children.Fri, 16 Nov 2012 09:12:12 ESThttp://www.sciencedaily.com/releases/2012/11/121116091226.htmArthritis study reveals why gender bias is all in the geneshttp://www.sciencedaily.com/releases/2012/11/121115210541.htm Researchers have pieced together new genetic clues to the arthritis puzzle in a study that brings potential treatments closer to reality and could also provide insights into why more women than men succumb to the disabling condition.Thu, 15 Nov 2012 21:05:05 ESThttp://www.sciencedaily.com/releases/2012/11/121115210541.htmClass of RNA molecules protects germ cells from damagehttp://www.sciencedaily.com/releases/2012/11/121115172255.htm Passing one's genes on to the next generation is a mark of evolutionary success. So it makes sense that the body would work to ensure that the genes the next generation inherits are exact replicas of the originals. Biologists have now identified one way the body does exactly that.Thu, 15 Nov 2012 17:22:22 ESThttp://www.sciencedaily.com/releases/2012/11/121115172255.htm

    Source: http://www.sciencedaily.com/rss/health_medicine/genes.xml

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    Thursday, November 29, 2012

    Interpretation

    Ladder or guardrails?Permissible, now there is a word subject to wide interpretation. Look at history or around the world or even on your own street. There is no question what one person finds permissible, another sees as offensive. For literally thousands of years, the way to take a good deal of the subjective-ness out of things is to make rules.

    In the world of residential construction these rules are known as building codes. Building codes themselves date back thousands of years. The Greeks and Romans created and used building codes. Archaeological records of a building being constructed by the Greek philosopher Socrates in part state:

    "He shall set the joints against each other, fitting, and before inserting the dowels he shall show the architect all the stones to be fitting, and shall set them true and sound and dowel them with iron dowels, two dowels to each stone?"

    Bowed deck main support beamThis I think demonstrates that it has long been understood when a building or structure is constructed, it must have a uniform method. It also must be strong and above all else, safe. It also shows incompetence and shoddy work are just as old as civilization or there would be less of a need for codes. Yet codes are only half the process. While they do exist, it does not necessarily mean they are followed by all. Another thing that hasn't changed with civilization's history. Thus the need for over site.

    The process used today in many parts of this country, not all, but many, is the permitting and inspection of construction. Basically permits are taken out with the local building department. Then at some point the new construction is inspected by the local building official. When the project meets his or her approval, the permit is closed.

    Let me repeat, the permit is closed.

    As a home inspector I am inform often that permits were pulled for _____. My response is always the same, "are they closed?" This can at times receive a quizzical look on the face of the one who made the statement.

    Four nails "securing" beam to postClosed?

    Recently I inspected a permitted deck. I really have come to despise decks. There is almost always something wrong.

    The first glaring defect was the ladder like guard rails and lack of handrails. Stooping to look under the deck at the support structure, I was dumbfounded by what I saw. The main support beam was very noticeably bowing. The reason was very clear, this beam by my estimate is well over 20 feet in length and devoid of an center support. A 4 x 4 post was stationed at each end, nothing any where in between. But wait, it gets better.

    Going around to one post of interest, I saw the pier was not squarely on the footing. Looking at the attachment to the beam, I saw four nails. Same on the back side. The support beam was actually two separate boards face nailed to the two posts. The center of this large deck in essence is supported solely on a total of 16 nails.

    Post not centered on bracket / footingThe best method for constructing a pier beam support is to set the beam on top of the posts. In this way the load is distributed evenly along the beam, with the piers fully supporting the beam, not a set of fasteners.

    There were several other issues with this permitted deck, which goes to show, permissible is open to interpretation, especially when circumventing the rules.

    ?

    James Quarello
    Connecticut Home Inspector
    Former SNEC-ASHI President
    NRSB #8SS0022
    JRV Home Inspection Services, LLC

     ASHI Certified Inspector

    To find out more about our other high tech services we offer in Connecticut click on the links below:

    Serving the Connecticut Counties of Fairfield, Hartford, Middlesex, New Haven, Southern Litchfield and Western New London.

    ?

    Source: http://activerain.com/blogsview/3533409/interpretation

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